Preliminary scientific studies reveal conolidine may inhibit unique ion channels, decreasing neuronal excitability and restricting ache signals. This mechanism is particularly applicable in neuropathic ache, in which abnormal signaling triggers persistent distress. Also, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to pain perception. Computational ... https://ahmadb380tkm0.goabroadblog.com/profile